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Polio:     more books (100)

Jonas Salk: Discoverer of the Polio Vaccine (People to Know) by Carmen Bredeson, 1993-08 The Story of Jonas Salk and the Discovery of the Polio Vaccine (Cornerstones of Freedom) by Jim Hargrove, 1990-08 Polio (Epidemics) by Allison Stark Draper, 2001-02 Triple Polio Challenge! [caption title]. by Duon H. Miller, 1955 Polio - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References by ICON Health Publications, 2004-02-13 Sister Elizabeth Kenny: Maverick Heroine of the Polio Treatment Controversy Polio and Me: In Nigeria and Malawi, Belgium, England and Other Places, 1955-1998 by Kenneth James Barnes, 1998-12 Polio (Diseases and People) by Alvin Silverstein, Virginia B. Silverstein, et all 2001-07 Polio's Legacy by George Gottfried, 1996-03-19 The Kenny Concept of Infantile Paralysis and Its Treatment. In collaboration with Sister Elizabeth Kenny. With a foreword by Frank R. Ober, MD by John F. POHL, 1943 Protection From Polio and Animal Research by Mira Louise, 1956 Jonas Salk and the Polio Vaccine (Unlocking the Secrets of Science) by John Bankston, 2001-08-10 patenting the Sun: Polio and the Salk Vaccine by Jane S. Smith, 1990 Polio Case Study by D. Male, B. Davey, 2009-05-23

lists with details

81. La Polio Et Son Vieillissement
    Article paru dans le n 430 de Kin sith rapie Scientifique.  
    http://www.post-polio.fr/BG_fichiers/lapolioetsonvieillissement.pdf

82. HANDICAP_POST_POLIO_ACCUEIL
    Propose des informations, des t moignages et des r flexions sur la vie de personnes handicap es par les s quelles de cette maladie, ou de leur entourage.  
    http://www.post-polio.fr/

83. Albert Sabin, MD, Oral Polio Vaccine Discovery, Cincinnati Children's Hospital M
    Information on Albert B. Sabin, MD, developer of the polio vaccine from the Cincinnati Children s Hospital Medical Center.  
    http://www.cincinnatichildrens.org/about/history/sabin.htm
    
    Extractions: Dr. Albert Sabin's Research Oral Polio Vaccine Discovery "Sabin Sunday" Sabin Education Center Born in Poland in 1906, Albert Sabin, MD, and his family left in 1921 to escape anti-Semitism. He came to Cincinnati Children's Hospital, Ohio, in 1939. Dr. Sabin's research, documented in some 350 scientific papers, would include work on pneumonia, encephalitis, toxoplasmosis, viruses, sandfly fever, dengue and cancer. But his passion was poliomyelitis, and this was where he turned his attention after World War II. Dr. Sabin first thought the polio virus gained entrance through the respiratory tract, then found evidence that entry was through the digestive system. His studies of incidence showed that, contrary to many diseases, acute polio was rare in urban populations with poor sanitation conditions (as existed in many parts of China). Return to Top Follow-up studies showed children in such places had protective antibodies in their blood, though they exhibited no sign of ever having had polio. Dr. Sabin reasoned such immune children had either contracted the viral infection as infants (when they had partial immunity transferred from their mothers) or been infected by an attenuated or weakened strain of the virus, which had produced immunity without acute symptoms.

84. Asociace Polio
    H jen a prosazov n z jm osob posti en ch n sledky poliomyelitidy  
    http://www.polio.cz

85. StandProud (formerly: IPVRC International Polio Victims Response Committee)
    From the International polio Victims Response Committee. Provides illustrations and photographs.  
    http://www.ipvrc.org/musclecontractures.htm
    
    Extractions: Can Make ... About "Muscle Contractures" Polio paralyzes motor nerves (most commonly those of the legs, but also frequently of the back and sometimes of the arms). The muscles which the paralyzed nerves are supposed to control do not get sufficient exercise and thus begin to atrophy soon after the patient has recovered from the active phase of the disease. When, as is often the case, the muscles atrophy unevenly on the two opposing sides of a joint, it is called "muscle imbalance" and, if left uncorrected, usually results in the joint coming to favor strongly a particular position (the stronger muscle pulling harder than the more paralyzed one). For example, often in polio cases, the muscles in the back of the thigh are less paralyzed and retain more strength (atrophy less) than the muscles toward the front of the thigh. Since the back thigh muscles which pull the leg back into a bent position meet little resistance from the front thigh muscles (used to pull the lower leg forward into the straight-leg position), the leg comes to "prefer" a bent position and the back thigh muscles become accustomed to not being fully stretched out very often. Over time, these muscles actually shrink or "contract." It becomes impossible to pull the leg, even manually with force, into the straight position anymore, and the muscles begin to pull tightly on the tendon behind the knee. A leg which can no longer be straightened out at the knee is said to have a "knee contracture."

86. PTU - Landsforeningen Af Polio-, Trafik- Og Ulykkesskadede: Forside
    Interesseforening, med medlemstilbud, forskning, nyheder, informationsmateriale og projekter.  
    http://www.ptu.dk/

87. Jaipurfoot.org :: Bhagwan Mahaveer Viklang Sahayata Samiti, Jaipur
    Provides free artificial limbs, jaipur foot, polio calipers, crutches, prosthesis to amputees and disabled in India and abroad.  
    http://www.jaipurfoot.org/

88. Polio    [www.gezondheid.be]   
    Dossier over kinderverlamming van gezondheid.be  
    http://www.gezondheid.be/index.cfm?fuseaction=dossier&art=86

89. Ipol (Poliovirus Vaccine Inactivated) Drug Information: User Reviews, Side Effec
    Full prescribing information from RxList.  
    http://www.rxlist.com/ipol-drug.htm#ids

90. Why PPS Is So Hard To Understand
    of muscle types, muscles indirectly affected by PPS, how PPS differs from ALS, immune vs autoimmune.  
    http://www.skally.net/ppsc/und.html
    
    Extractions: From: "Eddie Bollenbach" One of the reasons PPS is so difficult to get a handle on is that the physician cannot detect increases in weakness by manual muscle testing. Actually, I'm starting to think that whether you call it PPS or PPMA In a normal individual there are (broadly) two types of muscle fibers that are components of voluntary muscle: Type I and Type II fibers. They are also called "slow twitch (Type I)" and "fast twitch (Type II"). When you stain a muscle biopsy and look at it under the microscope the slow twitch Type I appear lighter in color while the faster Type II generally are darker. Again, in normals there is a random and approximately equal distribution of slow twitch and fast twitch fibers within the muscle. When you contract a muscle to do work the fast twitch contract first and have a high capacity for extracting energy from sugar anaerobically. That means even though you can't deliver oxygen rapidly to the muscle the fast twitch can do its thing, quickly, way before circulation can deliver oxygen. The result of this is a build up of lactic acid. When you initially contract a muscle to do work it is the type II's that contract. Anyway, If you had polio you probably have more slow twitch than fast twitch. For some reason now we are losing some of the slow twitch fibers we built up during recovery from acute polio. The result is an extreme fatiguability of muscles that used to work just fine. So we are having an inability to sustain muscle contraction. But instantaneous strength frequently remains intact.

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